Equine Lameness for the Layman. G. Robert Grisel, DVM

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Equine Lameness for the Layman - G. Robert Grisel, DVM

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more of the other limbs. Referred gait deficits are not genuine and disappear pursuant to successful treatment of the primary lameness. Example: A horse might exhibit a primary left hind weight-bearing lameness with a referred left front weight-bearing component. Intermittent Lameness A lameness that appears to come and go from one day to the next. Issues that occasionally provoke biomechanical interference can generate intermittent lameness, as can sporatic bouts of muscle cramping or inflammation. Example: Equine Hyperkalemic Periodic Paralysis (HYPP), a muscular disease caused by an inherited genetic mutation, can produce symptoms of intermittent lameness in Quarter Horses and related breeds. Shifting Lameness A lameness that appears to move from one limb or region of the horse to another, depending on the day or time of evaluation. Example: Some horses with bilateral forelimb navicular pain will exhibit shifting-limb lameness; one minute they’ll be worse in the left front and the next minute they’ll be worse in the right front. Metabolic Lameness Lameness that occurs as a result of something not directly related to the horse’s musculoskeletal or nervous system. Colic, pleuropneumonia, and aortic-iliac (“saddle”) thrombosis are metabolic ailments that can affect a horse’s stride and/or make the horse reluctant to move. Example: My mare exhibited severe metabolic lameness during her recent bout of pneumonia; she refused to walk down the barn aisle. Pathognomonic Lameness Produces one or more gait deficits unique to a specific pathologic condition. Since pathognomonic deficits are definitive, their detection enables the observer to make a cursory diagnosis of the horse’s problem. Example: “Goose-stepping” of the hind limb is considered to be pathognomonic for fibrotic semitendinosis myopathy.

      7 Primary versus Secondary Lameness

      Lameness comes in two basic forms: primary and secondary. Primary asymmetry occurs as a consequence of an event that originates independent of other preexisting lameness. Trauma, breed, age, and poor conformation could all play a role in the development of primary lameness. Identifying the primary gait deficit(s) is the chief goal of the veterinary examiner, because this is where treatment will eventually be directed.

      Secondary lameness, on the other hand, manifests as a consequence of one or more preexisting gait deficits elsewhere in the horse. It can be genuine (as in cases of associated and compensatory issues) or artificial (as in the case of referred asymmetry).

      The relationship between primary and secondary lameness is unidirectional (fig. 7.1). Secondary lameness would not exist without the presence of a primary underlying problem. This is an important concept when considering the fact that permanent resolution of secondary lameness would, at least in part, demand mitigation of its primary counterpart. As long as a primary problem exists, the potential for secondary pathology is not far behind.

       7.1 Primary and Secondary Lameness

      The relationship between primary and secondary lameness is unidirectional.

      On the other hand, secondary components may or may not coexist with primary lameness. A single primary lameness with no secondary elements would be classified as simple. Simple lameness is the most basic form since examination, diagnosis, and treatment are all directed toward a single anatomic region of the horse.

      In cases of complicated lameness, however, more than one anatomic region is involved. Differentiating regions that require preferential attention from those that don’t is one of the primary objectives of the adept observer. All primary issues will require accurate diagnosis and treatment in order to reestablish the horse’s performance. Depending on the duration and nature of secondary lameness, however, exclusive treatment may or may not be necessary. In many instances, secondary issues will spontaneously resolve once the primary issue has been successfully addressed. The smarter approach, therefore, is to identify and treat primary problems first.

      Consider the analogy of your car’s front-end alignment and how it affects tire wear. Poor alignment would be considered a primary issue. Accelerated tire wear would be expected to occur secondary to poor alignment. The expensive and time-consuming application of new tires might improve your car’s performance in the short term, but the persistence of poor alignment will repeatedly result in premature tire depletion. The appropriate course of action is unidirectional: fix the car’s alignment first, then evaluate the status of the tires to determine if and when replacement is necessary. Successful management of a horse’s soundness over the long term requires a similar approach.

      Those of us that regularly and carefully observe our horses in motion will see fewer complicated cases because we are more likely to detect gait abnormalities soon after their onset and before additional primary or secondary issues have time to develop. Unless it’s due to a common traumatic event, it is relatively rare for two separate, unrelated problems to occur simultaneously. In this way, regular observation actually simplifies the process of evaluation by making the incidence of multifactorial and secondary lameness less probable.

      Even so, the majority of lameness cases are complicated. In order to orchestrate our diagnostic and treatment plans appropriately, we must first recognize and designate each component of the horse’s lameness as primary or secondary. Although this task may seem daunting to the casual observer, there are some basic guidelines that will help. Knowledge of the typical ways that secondary lameness manifests at the trot can aid in accurate classification.

      Consider the following points:

       All lame horses have at least one primary component that is contributing to their altered movement. That said, most lame horses have only one primary component. Multifactorial lameness is a relatively uncommon form of complicated lameness.

       There is little correlation between the degree of lameness and its primary/secondary denotation. A severe lameness does not certify a “primary” classification, since secondary lameness is often more visibly obvious than its underlying primary inciter.

       Forelimb lameness is more likely to be secondary to hind limb lameness than vice versa. Since the forelimbs normally experience the majority (approximately two-thirds) of the horse’s weight, very slight hind limb lameness may be enough to significantly influence forelimb load and action. By contrast, marked forelimb lameness is typically required to generate corresponding repercussions behind.

       When forelimb and hind limb asymmetries coexist:If the hind limb component is worse, it is more likely to be primary.If the forelimb component is considerably worse (by more than one grade), it is more likely to be primary.If the severity of the two is comparable (within one grade of each other), the hind limb component is more likely to be primary.

       In my opinion, back and neck issues are more likely to be secondary to limb lameness than vice versa. In fact, secondary sacroiliac (SI) and back pain are anticipated consequences of chronic and/or moderate hind limb lameness in the quadruped.

      Compensatory lameness occurs as

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