however, largely because we didn’t understand why aging occurs. Up until very recently, the best thing we had was a list of aging hallmarks. The Information Theory of Aging could change that.
There is nothing wrong with using the hallmarks to guide interventions. We can probably have a positive impact on people’s lives by addressing each of them. It’s possible that interventions aimed at slowing telomere deterioration will improve people’s long-term well-being. Maintaining proteostasis, preventing deregulation of nutrient sensing, thwarting mitochondrial dysfunction, stopping senescence, rejuvenating stem cells, and decreasing inflammation might all be ways to delay the inevitable. Indeed, I work with students, postdocs, and companies around the globe that are developing solutions to each one of these hallmarks and hope to continue.23 Anything we can do to alleviate suffering we should do.
But we’re still building nine dams on nine tributaries.
In coming together to tackle the “new science of aging,” as the attendees of the Royal Society meeting termed this fight in their 2010 meeting, increasing numbers of scientists are starting to acknowledge the possibility and potential inherent in heading upstream.
Together we can build a single dam—at the source. Not just intervene when things go wrong. Not just slow things down. We can eliminate the symptoms of aging altogether.
This disease is treatable.
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