A Clinical Guide to Urologic Emergencies. Группа авторов

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Benson, D.A., Stockinger, Z.T., and Mcswain, N.E. (2005). Embolization of an acute renal arteriovenous fistula following a stab wound: case report and review of the literature. Am. Surg. 71 (1): 62–65.

      93 93 Uflacker, R., Paolini, R.M., and Lima, S. (1984). Management of traumatic hematuria by selective renal artery embolization. J. Urol. 132 (4): 662–667.

      94 94 Reilly, K.J., Shapiro, M.B., and Haskal, Z.J. (1996). Angiographic embolization of a penetrating traumatic renal arteriovenous fistula. J. Trauma 41 (4): 763.

      95 95 Tucci, P., Doctor, D., and Diagonale, A. (1979). Embolization of post‐traumatic renal arteriovenous fistula. Urology 13 (2): 192–194.

      96 96 Reigle, M.D., Selzman, A.A., Elder, J.S., and Spirnak, J.P. (1998). Use of ureteral stents in the management of major renal trauma with urinary extravasation: is there a role? J. Endourol. 12 (6): 545–549.

      97 97 Buckley, J.C. and McAninch, J.W. (2006). Selective management of isolated and nonisolated grade IV renal injuries. J. Urol. 176 (6 Pt 1): 2498–2502; discussion 2502.

      98 98 Heyns, C.F. (2004). Renal trauma: indications for imaging and surgical exploration. BJU Int. 93 (8): 1165–1170.

      99 99 Moudouni, S.M., Patard, J.J., Manunta, A. et al. (2001). A conservative approach to major blunt renal lacerations with urinary extravasation and devitalized renal segments. BJU Int. 87 (4): 290–294.

      100 100 Husmann, D.A. and Morris, J.S. (1990). Attempted nonoperative management of blunt renal lacerations extending through the corticomedullary junction: the short‐term and long‐term sequelae. J. Urol. 143 (4): 682–684.

      101 101 Keihani, S., Anderson, R.E., Fiander, M. et al. (2018). Incidence of urinary extravasation and rate of urethral stenting after high‐grade renal trauma in adults: a meta‐analysis. Transl. Androl. Urol. 7 (Suppl. 2): S169–S178. https://doi.org/10.21037/tau.2018.04.13.

      102 102 Prakash, S.V., Mohan, C.G., Reddy, V.B. et al. (2015). Salvageability of kidney in Grade IV renal trauma by minimally invasive treatment methods. J. Emerg. Trauma Shock 8 (1): 16–20. https://doi.org/10.4103/0974‐2700.145418.

      103 103 Keller, M.S., Eric Coln, C., Garza, J.J. et al. (2004). Functional outcome of nonoperatively managed renal injuries in children. J. Trauma 57 (1): 108–110; discussion 110.

      104 104 Keller, M.S. and Green, M.C. (2009). Comparison of short‐ and long‐term functional outcome of nonoperatively managed renal injuries in children. J. Pediatr. Surg. 44 (1): 144–147; discussion 147.

      105 105 Tasian, G.E., Aaronson, D.S., and McAninch, J.W. (2010). Evaluation of renal function after major renal injury: correlation with the American Association for the Surgery of Trauma Injury Scale. J. Urol. 183 (1): 196–200.

      106 106 Wessells, H., Deirmenjian, J., and McAninch, J.W. (1997). Preservation of renal function after reconstruction for trauma: quantitative assessment with radionuclide scintigraphy. J. Urol. 157 (5): 1583–1586.

      107 107 McGonigal, M.D., Lucas, C.E., and Ledgerwood, A.M. (1987). The effects of treatment of renal trauma on renal function. J. Trauma 27 (5): 471–476.

      108 108 Watts, R.A. and Hoffbrand, B.I. (1987). Hypertension following renal trauma. J. Hum. Hypertens. 1 (2): 65–71.

      109 109 Lebech, A. and Strange‐Vognsen, H.H. (1990). Hypertension following blunt kidney injury. Ugeskr. Laeger 152 (14): 994–997.

      110 110 Meyrier, A., Rainfray, M., and Lacombe, M. (1988). Delayed hypertension after blunt renal trauma. Am. J. Nephrol. 8 (2): 108–111.

      111 111 Chedid, A., Le Coz, S., Rossignol, P. et al. (2006). Blunt renal trauma‐induced hypertension: prevalence, presentation, and outcome. Am. J. Hypertens. 19 (5): 500–504.

      112 112 Montgomery, R.C., Richardson, J.D., and Harty, J.I. (1998). Posttraumatic renovascular hypertension after occult renal injury. J. Trauma 45 (1): 106–110.

      113 113 Goldblatt, H., Lynch, J., Hanzal, R.F., and Summerville, W.W. (1934). Studies on experimental hypertension: I. The production of persistent elevation of systolic blood pressure by means of renal ischemia. J. Exp. Med. 59 (3): 347–379.

      114 114 Fuchs, M.E., Anderson, R.E., Myers, J.B., and Wallis, M.C. (2015). The incidence of long‐term hypertension in children after high‐grade renal trauma. J. Pediatr. Surg. 50 (11): 1919–1921.

      115 115 von Knorring, J., Fyhrquist, F., and Ahonen, J. (1981). Varying course of hypertension following renal trauma. J. Urol. 126 (6): 798–801.

      116 116 Working Group on Renovascular Hypertension (1987). Detection, evaluation, and treatment of renovascular hypertension. Final report. Arch. Intern. Med. 147 (5): 820–829.

       Jonathan Wingate

       Madigan Army Medical Center, Tacoma, WA, USA

      The World Health Organization (WHO) defines traumatic injuries as either intentional (interpersonal violence related, war‐related, or self‐inflicted injuries) or unintentional injuries (motor vehicle collisions, falls, etc.). Traumatic injuries are the leading cause of death in the United States for people aged 1–44 years [1]. Worldwide, traumatic injuries are the ninth leading cause of death and disproportionately affects males and those in low and middle‐ income countries (LMIC) [2]. By 2030, the WHO projects a 28% increase in global deaths due to trauma and injury [3].

      Civilian Versus Military Trauma

      In civilian trauma, the kidneys are the most commonly injured genitourinary (GU) organ. The kidneys are injured in 1–5% of trauma patients and comprise up to 24% of traumatic solid abdominal organ injuries [4–6]. Stratifying by mechanism, there is wide geographical variation for penetrating renal injury (PRI) versus blunt renal injury (BRI) and the reported range for PRI is between 10.9 and 43.9% of all renal injuries [7–9].

      Historically, in wartime trauma, the kidneys were the predominant GU organ injured during conflicts in the early and mid‐twentieth century. Hugh Hampton Young described the GU injury patterns for Allied Forces in World War I and noted a 7.3% incidence of renal trauma at time of laparotomy with a 50% mortality rate [10]. These were almost all penetrating injuries, with 93.9% of soldiers having a concomitant hollow viscous injury. Surprisingly, the nephrectomy rate was only 18.1% [10]. There has been a paradigm shift in GU injuries due to advancements in technology – specifically the use of Kevlar body armor – resulting in a significant decline of PRIs and an increase in complex lower tract blast injuries, the signature GU injury of the recent conflicts in the Middle East [11–14].

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